Cardiology Questions

During hypertensive left ventricular hypertrophy, what does the ventricular wall stiffness impair and what does this cause

What are the poor prognostic indicators for Tachyarrhythmia?

Depends on the cause of anemia. Involves replacement of missing nutrients needed for the production of hemoglobin and proper red blood cell development

Blocks Angiotensin II from attaching to receptor sites and causing vasoconstriction. Reduces blood pressure

Interrupt Vitamin K production of cofactors that help blood to clot. Decrease Vit. K in the body. Block cofactors thrombin and fibrin that are required to form clots

Dilates arteries and decreases heart rate. Results in decreased blood pressure

IFU: Congestive Heart Failure (CHF) => increase blood volume causing workload on the heart

- Microzide (hydrochlorothiazide)- Aldactone (spironolactone)- Lasix (furosemide)

- Nitrostat (nitroglycerin)- Nitrolingual (nitroglycerin)

Relaxes smooth muscle around blood vessels. Dilates arteries/veins. Decreases O2 used by cardiac muscle

- Amicar (aminocaproic acid)- Cyklokapron (tranexamic acid)

- Apresoline (hydralazine)- IMDUR (isosorbide mononitrate)

- Diovan (valsartan)- Benicar (olmesartan)

Deactivates the NA+/K+ ATPase pump and activates the Na+/Ca+ exchanger. Results in increased Ca+ inside of the cell = improved heart contractions, circulation, rhythm and promotes removal of excess fluid by kidneys

Increases urinary excretion of Na+, H2O, and sometimes K+. Less fluid in the body helps reduce blood pressure. +There are 3 subclasses*

Relaxes smooth muscles of blood vessels. Dilates arteries/veins = decreased resistance on the heart making it easier to pump

Block potassium channels on cardiac muscle cells and keeps potassium from leaving the cells. Increased potassium inside of cells = normal heart rhythm

Works on kidneys to prevent conversion of Angiotensin I to Angiotensin II. Decreases Na+ and H2O retention => decreased blood pressure

Inhibits the secretion of triglycerides from the liver. Reduces blood triglyceride levels and activates fat enzymes (lipoprotein lipase) to aid with the breakdown of fat

Slows down the liver's production of cholesterol by blocking HMG-CoA reductase enzyme in the liver